A study showed that lncRNA MALAT-1 is upregulated in DLBCL cell lines, and the downregulation of MALAT-1 inhibits the proliferation and migration of DLBCL cells, induces a cell cycle arrest, activates autophagy, and blocks doxorubicin-induced epithelial–mesenchymal transition, thereby reducing the doxorubicin resistance in DLBCL (10). Here, MALAT1 is linked to diffuse large B-cell lymphoma.