It had been demonstrated that colorectal cancer–associated transcript 1-long isoform (CCAT1-L) as a super-enhancer RNA (a cluster of eRNA) could bind at MYC to induce many pro-proliferative genes and maintain cancer cell survival by exempting from immune surveillance and the antitumor immune response (Cotterman et al., 2008; Kim et al., 2017). This evidence concerns the gene MYC and cancer.