Interestingly, when analyzing these eCB members in monocytes, the immunophenotyping not only revealed that CB2 expression was significantly reduced in AD patients compared to healthy controls (MFI = 334.40 ± 18.31 vs. 445.30 ± 30.38), with the levels of CB1 remaining unchanged (Figure 2C), but also that FAAH expression was significantly increased (MFI = 989.40 ± 64.53 vs. 742.30 ± 69.85). This evidence concerns the gene CNR2 and Alzheimer disease.