Novel research should be conducted in order to confirm a precise function of enhanced expression of GLUT4 in OC development and provide information on whether inhibition of GLUT 4 will have beneficial effects in transporter-targeted therapies in some types of OC associated with resistance to chemotherapy because some research shows overexpression of GLUT 4 in mucinous and clear cell adenocarcinomas [65]. This evidence concerns the gene SLC2A4 and clear cell adenocarcinoma.