Using a different approach, autophagy, which is an essential part of tumor metabolism that protects ovarian CAFs against oxidative stress, was also found to be a potential therapeutic option as targeting maternally expressed 3 (MEG3) and metastasis associated lung adenocarcionoma transcript 1 (MALAT1) lncRNAs that modulate autophagy can potentially diminish CAF and ovarian cancer progression [25]. The gene discussed is MEG3; the disease is ovarian carcinoma.