Since we previously observed diminished allergen-specific IgA levels in eczema and asthma patients [12], and that aberrant allergen-specific and virus-specific IgA responses may even precede asthma development, future studies could investigate whether restoration of IgA production may help to protect asthma patients against viral infections and associated exacerbations [12,30]. The gene discussed is CD79A; the disease is asthma.