Finally, MM patients who were KIR3DS1+ (activating KIR encoding gene) experienced shorter PFS after autoHSCT and this was more evident in patients that were at complete or partial remission at transplantation and who lacked the ligand for the inhibitory KIR3DL1, i.e., HLA-Bw4 [130]. This evidence concerns the gene KIR3DL1 and Miyoshi myopathy.