Previously, CCL2/monocyte chemotactic protein-1 (MCP-1) was found to have higher levels of CCL2 and CCL5 (regulated on activation, normal T cells expressed, and secreted (RANTES)) in the BALF, associated with the development of status asthmaticus, along with increased IL-5, compared with patients with mild asthma [34]. The gene discussed is CCL2; the disease is asthma.