Low SES, for example, has been associated with inflammatory and diabetic genes such as TLR3 [117], NLRP12 [118], F8 [119], KLRG1 [120], CD1D [121], as well as the stress-associated genes OXTR [122], FKBP5 [123], and AVP [124], suggesting, as we have previously proposed, that a negative early life environment acts through inflammatory pathways that are also associated with T2D, targeting pathophysiological factors such as stress and inflammation, and participating in the aetiopathology of T2D [108]. The gene discussed is KLRG1; the disease is type 2 diabetes mellitus.