Previous studies on CRP as a risk marker for STEMI patients were mostly focused on clinical endpoints other than HF and had several methodological limitations such as a lack of treatment with percutaneous coronary intervention (PCI) and evidence-based pharmacotherapies in all patients, CRP measurement at a single time point, no use of high-sensitivity CRP assays, and/or short-term follow-up (e.g., [12,18,19,35,36,37,38,39,40,41]). Here, CRP is linked to hydrops fetalis.