As glucose transporters (including GLUT1) are responsible for 18F-fluorodeoxyglucose (FDG) accumulation in PDA basal microvilli, structures that are not present in the microvasculature of normal tissues [128], the use of FDG positron emission tomography (FDG-PET) could predict outcomes and early recurrence after surgery in PDA patients [129]. Here, SLC2A1 is linked to Patent ductus arteriosus.