A recent study examined the retinal pathophysiology of LCA in a zebrafish model, the obelixtd15 (obetd15) mutant, which carries a missense mutation in kcnj13 (c.502T>Cp.[Phe168Leu]) and exhibits an ocular phenotype similar to that of LCA patients, albeit with a later onset [160]. Here, KCNJ13 is linked to Leber congenital amaurosis.