Indeed, the overexpression of miR-4429 in radio-resistant cells decreased the mRNA and protein levels of RAD51 recombinase (RAD51) (Table 2), a crucial regulator of DNA damage repair that had been previously reported as a contributor of radio and chemoresistance in cervical cancer cells [94], thus leading to increased cell death. The gene discussed is RAD51; the disease is cervical carcinoma.