AR and glioblastoma: A growing body of evidence also suggests AR as a modulator of treatment response in GBM, for example: (i) AR confers radiation resistance [73]; (ii) the nuclear translocation of AR is affected by cedrol treatment, suppressing GBM progression [74]; (iii) the 5α-reductase enzyme inhibitor dutasteride in combination with AR antagonists effectively decreased the proliferation of GBM cells [75]; and (iv) a curcumin analog induced the ubiquitination of AR and consequent degradation, suppressing the growth of temozolomide-resistant cells [76].