In pathogenic SARS-CoV-2 infection leading to ALI/ARDS, deregulated inflammation involves an increase in the levels of proinflammatory cytokines, several other inflammatory mediators (e.g., chemokines), and inflammatory transcription factors (such as nuclear factor kappa B and sterol regulatory element binding proteins (SREBPs)) [13]. This evidence concerns the gene CNBP and acute respiratory distress syndrome.