Given that FAK and paxillin expression is elevated in prostate cancer and both proteins are associated with tumor progression, lymph node metastasis, and/or shortened survival [58,59], it is also plausible that in our cancer model, the paxillin/FAK pathway could contribute to the regulation of Snail expression by ERK and JNK. The gene discussed is SNAI1; the disease is prostate carcinoma.