Similar results were observed in our present study using HEC-1-A and Ishikawa cells, where TGF-β1 treatment induced a low expression of E-cadherin and high expression of N-cadherin, vimentin, and α-SMA compared to control group, highlighting the role of TGF-β in induction of EMT in endometrial cancer. Here, ACTA1 is linked to endometrial cancer.