IFNG and chlamydia infectious disease: Our current findings revealed that FP-treated mice produced significantly higher amounts of IL-17A in response to C. pneumoniae infection, suggesting that this increase in IL-17A production might also result in the development of C. pneumoniae inhibition; as observed in a previous study, IL-17A can synergise with IFN-γ, playing a protective role in Chlamydia infection [42].