AMI patients (n = 125) had significantly higher systemic S100B levels, as compared to those in stable angina pectoris (n = 122) and controls subjects (n = 120) [median (IQR):302.16 (606.50–103.18) vs. 159.50 (315.69–45.38) vs. 119.45 (300.23–20.41)] (Figure 1A). This evidence concerns the gene S100B and angina pectoris.