CHIT1 and fungal infectious disease: Furthermore, neutrophilic recognition of the fungal pathogen C. albicans causes the release of anti-microbial molecules from granules at the site of fungal infection including the chitin digestion enzyme chitotriosidase (Chit1), which acts on the non-reducing end of the chitin microfibril, releasing copious N-acetyl-D-glucosamine (NADG) dimer, chitobiose, one by one from the chitin chain (13, 14).