NR3C1 and myocardial infarction: Meanwhile, prohibiting corticosterone signaling through cardiomyocyte-specific glucocorticoid receptor ablation results in increased cardiomyocyte proliferation and heart regeneration after myocardial infarction at postnatal day 778, although this result was not replicated in a study that treated mice with a glucocorticoid receptor antagonist after myocardial infarction at postnatal day 179.