FGF7 and osteomalacia: A previous study demonstrated that FGF7 is overexpressed in some patients with mesenchymal tumors associated with osteomalacia, renal phosphate wasting, and hypophosphatemia.[6] The investigators found that FGF7 in tumor cell cultures inhibited in vitro sodium-dependent phosphate transport in opossum kidney proximal tubule cells, and that this inhibitory activity of tumor-derived cultures was abrogated by coincubation with neutralizing anti-FGF7 antibodies.