In agreement with the changes in the severity of cardiac dysfunction after TAC (Figures 1A and 1B), the density of myocardial LYVE‐1+ and VEGFR‐3+ lymphatic capillaries and the LYVE‐1+ lymphatic‐to‐WGA+ CM ratio were both markedly increased in week 1, peaked in week 2, and then remarkably decreased in weeks 4–6 (Figures 1C and 1D). This evidence concerns the gene LYVE1 and persistent truncus arteriosus.