In agreement with the changes in the severity of cardiac dysfunction after TAC (Figures 1A and 1B), the density of myocardial LYVE‐1+ and VEGFR‐3+ lymphatic capillaries and the LYVE‐1+ lymphatic‐to‐WGA+ CM ratio were both markedly increased in week 1, peaked in week 2, and then remarkably decreased in weeks 4–6 (Figures 1C and 1D). The gene discussed is FLT4; the disease is persistent truncus arteriosus.