Another European study by Zaliova et al. revealed only 1/75 (1.3%) pediatric B-ALL case with ABL1 gene rearrangement [27] while ABL-class fusions were present in 40/1389 (2.9%) high-risk pediatric B-ALL patients in an US Children’s Oncology Group study [13]. This evidence concerns the gene ABL1 and precursor B-cell acute lymphoblastic leukemia.