COVID-19 triggers an exaggerated immune response in infected patients, and a variety of inflammatory cytokines, such as interleukin (IL)-1β, IL-6, IL-8, interferon (IFN)-γ, and chemokines, such as granulocyte colony stimulating factor (G-CSF), interferon gamma-induced protein 10 (IP-10), monocyte chemoattractant protein-1 (MCP-1), and macrophage inflammatory protein 1α (MIP-1α), were elevated in severe COVID-19 patients (24, 25). This evidence concerns the gene CCL2 and COVID-19.