The low T3 syndrome is commonly observed in several acute and chronic clinical conditions such as in sepsis [74], myocardial infarction [75], and chronic heart failure [76, 77], and is the result of an adaptive mechanism whose severity is related to the levels of systemic inflammation [78], serum circulating cortisol, and disturbance of peripheral thyroid hormone transport and metabolism [79]. The gene discussed is TG; the disease is myocardial infarction.