GSTM1 and malaria: The sickle‐cell trait condition, consequence of the heterodimerisation of a normal haemoglobin (Hb) A with an Hb presenting the β6 Glu → Val substitution (HbS), is well known to be associated with a relative protection from Pf infection, development of malaria symptoms and severe disease outcomes, but not other infections.7, 8, 9, 10, 11, 12, 13, 14