HNF1B and type 2 diabetes mellitus: We hypothesize that the induction of lnc13509 impacts various biological and pathological responses regulated by miR802. Elevated expression of miR802 is seen in type-II diabetes [73] and in livers of high fat diet-fed mice, where it impairs glucose metabolism by silencing the transcription factor HNF1B and by increasing oxidative stress [74–76].