When T. gondii infection disrupts this precarious balance in LTβR−/− mice, the dysregulation becomes more pronounced; on the one hand, LTβR−/− mice have lower levels of IFN-γ in the serum early during infection, but on day 10 p.i., when WT mice already show decreased IFN-γ levels, they remain high in LTβR−/− mice, not only in serum but also in the lungs. This evidence concerns the gene IFNG and infection.