Recently, a study on CFs isolated from mice hearts upon angiotensin II (Ang II)-induced cardiac fibrosis post-MI revealed that miR-30b-5p and miR-22-3p were downregulated, whereas the platelet activating factor receptor (PTAFR) was upregulated [X. S. (78)]. The gene discussed is PTAFR; the disease is myocardial infarction.