NFKB1 and gastric cancer: A possible mechanism to explain PD‐L1 expression by p62/SQSTM1 in gastric cancer has been the demonstration of NF‐κB activation, which impacts the PD‐L1 promoter.[23] We confirmed that that IRIN treatment can induce the phosphorylation of the p65 (p‐p65) subunit of NF‐κB in KPC cells (Figure S4, Supporting Information).