The exciting work shown included the first vertebrate tagging (with Dendra2) of a matrix molecule (Lamb1); a role for TTBK2 in regulating ADAMTS‐5‐mediated aggrecan degradation; an analysis of missense variants in COL4A1 and COL4A2 in patients with sporadic intracerebral haemorrhages; the use of atomic force microscopy to demonstrate the flexibility that interruption sequences provide type IV collagen; and an immuno‐matrix talk describing the recruitment of macrophages into kidney disease models. This evidence concerns the gene COL4A1 and intracerebral hemorrhage.