Furthermore, we performed CRISPR-Cas9 knockout of LINC01614 in MKN28, MKN1, and GES1 cells and observed attenuated cell proliferation, defects in colony formation, and significantly reduced migration in wound healing assay (Figs. 6E–H and S11), supporting the oncogenic role of LINC01614 in promoting GC cell growth and migration. Here, LINC01614 is linked to gastric cancer.