SEMA6B and melanoma: Similarly, the ectopic expression of miR-30b/-30d has been demonstrated to enhance the metastatic properties of melanoma cells by binding to GALNT7, while restoration of the expression of GALNT7 has been reported to reverse the results triggered by the upregulation of miR-30d,12 supporting the notion that SEMA6B contributes to the regulatory functions of miR-30b/-30d in the progression of GBC.