Our results were in line with this aforementioned evidence, which showed that cell apoptosis and inflammation (reflected by supernatant TNF-α, IL-1β, IL-6, and IL-17 levels) were decreased in miR-146a-inhibitor-treated cells compared with NC inhibitor-treated cells in both PC12 and cortical neuron cellular AD models. The gene discussed is IL1B; the disease is Alzheimer disease.