EIF5A2 and ovarian cancer: Our function rescue experiments showed that some deprived CSC properties induced by EIF5A2 knockdown, such as decreased expression of stem markers, inhibited spheroid forming ability, and reduced chemo-resistance and tumorigenicity, were partially reversed by KLF4 overexpression, suggesting that KLF4 participates in the process of EIF5A2 modulating stemness in ovarian cancer cells.