MALAT1 and Parkinson disease: As MPP+ could cause oxidative stress and mitochondrial dysfunction, thus injure dopaminergic neurons, leading to PD pathophysiology [28, 29], SK-N-SH and SK-N-BE cells were treated with MPP+ to establish the MPP+-stimulated cell model of PD, and we aimed to investigate the biological role and mechanism underlying MALAT1 in PD.