To gain a more in-depth understanding of the underlying mechanisms, we used a literature mining approach to identify cell adhesion molecules that are involved in MM progression, including the integrin family of proteins such as integrin β1 (ITGB1), β3 (ITGB3), β7 (ITGB7), α4 (ITGA4), α5 (ITGA5) and αV (ITGAV), E-cadherin (E-cad), N-cadherin (N-cad), and β-catenin (B-cat) [35–38]. The gene discussed is ITGA5; the disease is Miyoshi myopathy.