ACHE and myasthenia gravis: Chronic inflammation mediated by helper T cell 17 (Th17) cells, follicular Th cells promoting the production of autoantibodies by B cells and plasma cells, regulatory T (Treg) cell dysfunction activating immune response may be the causes of exacerbation of MG.[20] Although the standard treatment of myasthenia gravis includes steroids, acetylcholinesterase, rituximab, immunosuppressants and thymectomy, the number of relapses, and myasthenia gravis crises is still increasing.[21]