TNFSF4 and cytomegalovirus infection: Taken together, we conclude that neither gp68 nor gp34 individually are able to efficiently antagonize FcγR activation by IgG-opsonized viral antigens in the physiological context of HCMV infection (Figure 2), that is, in the presence of non-immune IgG mitigating their inhibitory potential (Figure 6B and Figure 6C).