Since FAK is upregulated in human NB cell lines and clinical samples, it has been suggested that FAK is important for NB progression and metastasis [7] and other malignancies, such as ovarian serous cystadenocarcinoma, breast invasive carcinoma, and colorectal adenocarcinoma [8,9]. The gene discussed is PTK2; the disease is ovarian serous cystadenocarcinoma.