This is in accordance with the aforementioned report showing enhanced response of BMDMs to IL-4 following Wnt3a treatment via activation of STAT3 in a model of kidney injury (Feng et al., 2018), as well as with the suggestion that STAT3 and STAT6 cooperate to increase Arg1 gene expression (reviewed in Pourcet and Pineda-Torra, 2013). The gene discussed is ARG1; the disease is kidney injury.