In an experimental model of acute respiratory distress syndrome (ARDS) induced by Pseudomonas aeruginosa (P. aeruginosa) infection or aerosolized lipopolysaccharide (LPS), pulmonary ILC3s play a crucial role in recruiting neutrophils to the lung, associated with myeloperoxidase (MPO), IL-6 and the IL-17-dependent neutrophil chemoattractant MIP-2 (Muir et al., 2016). This evidence concerns the gene CXCL2 and acute respiratory distress syndrome.