Likewise, while a specific role for FARSA in cancer has not been identified, dysregulation of aminoacyl tRNA synthetases contribute to initiation, maintenance, and progression of carcinogenesis (52). Since the rate of cell growth has been demonstrated to be proportional to ribosomal biogenesis (53), it is likely that increased midasin and altered ribosomal protein expression could prime the translational machinery for increased protein synthesis or stabilize key proteins involved in cell proliferation. The gene discussed is FARSA; the disease is cancer.