ACTA1 and chronic kidney disease: Concurrently, the fibrous cap VSMC (α‐SMA+CD31−) contents in CKD/ApoE−/− mice were much less than those in Sham/ApoE−/− mice (Figure 2A,C; Figure S1H,I, Supporting Information), suggesting that VSMC loss might contribute to plaque vulnerability in CKD/ApoE−/− mice.