While it is possible that AHR and/or ASB2 may be modulating NK cell survival in the tumor instead of migration, our previous extensive analysis of conventional NK cells within AhR-deficient mice did not reveal any appreciable differences in the numbers of conventional NKp46+NK1.1+CD3−CD19− NK cells in the spleens of AhR-deficient mice compared with WT littermates (18). The gene discussed is AHR; the disease is neoplasm.