The primary findings of this study were: 1) the divergent profiles of subsequent risk amongst the different phenotypes of myocardial injury, 2) additive risk for subsequent MI in repeat presentations of acute myocardial injury with recognized CAD, 3) the value of troponin as a cardiac-selective risk stratification tool in patients with high burden of competing non-cardiac comorbidities, and 4) the high proportion of non-MI hs-cTnT elevations. Here, TNNT2 is linked to coronary artery disorder.