Jin et al. showed that commensal bacteria, through Myd88 dependent signaling, induced IL-17 producing Vγ6+Vδ1+γδ T cells in genetically engineered KP (KrasLSL-G12D; p53flox/flox) models of lung adenocarcinoma, thus leading to an immunosuppressive TME. The gene discussed is IL17A; the disease is lung adenocarcinoma.