In parallel, development of midbrain sections from individuals with the diagnoses of dementia with Lewy bodies, of non-PRKN-linked, sporadic PD as well as of cases with incidental Lewy bodies readily demonstrated eDAB reactivity overlapping with neuromelanin for all three anti-parkin clones (Supplementary Fig. 9d–g, online resource; and data not shown). The gene discussed is PRKN; the disease is Parkinson disease.