To test this hypothesis and prioritize genes involved in tumor development that could be regulated by SALL2, we compared genes nearby H3K27ac sites opened upon overexpression of POU3F2 + SOX2 + SALL2 in differentiated glioblastoma cells (DGC) against SALL2 targets in MGG8TPC cells, both datasets already reported by Suvá et al. (2014) (designated as Novel TSS H3K27ac). This evidence concerns the gene SALL2 and glioblastoma.