SALL2 and neoplasm: To test this hypothesis and prioritize genes involved in tumor development that could be regulated by SALL2, we compared genes nearby H3K27ac sites opened upon overexpression of POU3F2 + SOX2 + SALL2 in differentiated glioblastoma cells (DGC) against SALL2 targets in MGG8TPC cells, both datasets already reported by Suvá et al. (2014) (designated as Novel TSS H3K27ac).